Postdoctoral Scholars
About BELINDA
I was an MD/PhD student at Harvard and received my PhD in the laboratory of Bill Hahn where I leveraged genome-scale perturbation screens to understand molecular pathways that mediate vulnerability and resistance to targeted chemotherapy in cancer cells. I completed adult psychiatry residency at UCSF and stayed for child and adolescent psychiatry fellowship. For my post-doctoral training, I am interested in using using systems biology approaches to study risk genes identified by human genomic studies in order to understand the pathobiology underlying childhood-onset psychiatric disorders.
AWARDS
Sorensen Foundation Fellowship in Child and Adolescent Fellowship (2022-2025)
UCSF Weill Clinician Scientist Award (2023) NIMH U01 MD/PhD Administrative Supplement (2021-2023) NIMH Outstanding Resident Award (2020) SIGNIFICANT PUBLICATIONS
Willsey HR, Willsey AJ, Wang B, State MW. Genomics, convergent neuroscience, and progress in understanding autism spectrum disorders. Nature Reviews Neuroscience. 2022.
Wang S, Wang B, Drury V, Drake S, Sun N, Alkhairo H, Arbalaez J, Duhn C, Tourette International Collaborative Genetics (TIC Genetics), Heiman GA, Tischfield JA, Fernandez TV, O’Donovan MC, Thapar A, State MW, Jeremy AJ. Rare coding variants on chromosome X contribute to male sex bias in autism, Tourette syndrome, and attention-deficit/hyperactivity disorder. medRxiv. 2022. Searles Quick VB*, Wang B*, State MW. Large genomic data approaches to understand etiology of and develop treatment for autism spectrum disorder. Neuropsychopharmacology, 2020. Wang B*, Krall EB*, Aguirre AJ*, Kim M, Widlund HR, Doshi MB, Sicinska E, Sulahian R, Goodale A, Cowley GS, Piccioni F, Doench JR, Root DE, Hahn WC. ATXN1L, CIC, and ETS transcription factors modulate sensitivity to MAPK pathway inhibition. Cell Reports, 2017. Krall EB*, Wang B*, Munoz DM*, Ilic N, Raghavan S, Niederst MJ, Yu K, Ruddy DA, Aguirre AJ, Kim JW, Redig AJ, Gainor JF, Williams JA, Asara JM, Doench JG, Janne PA, Shaw AT, McDonald Iii RE, Engelman JA, Stegmeier F, Schlabach MR, Hahn WC. KEAP1 loss modulates sensitivity to kinase targeted therapy in lung cancer. Elife, 2017. *Co-first authors FUN QUESTION: if you could be any animal, what would it be and why?
I would be an octopus because they are intelligent and resilient, and they have eight arms to work with (which can regenerate!). |
about sHENG
I received my Ph.D. from China Agricultural University & National Institute of Biological Sciences, Beijing, where I worked on postzygotic variant analysis from either healthy donors or rare disease patients using whole-genome sequencing, whole-exome sequencing, single-cell sequencing, and other related sequencing methods. During my postdoc training, I mainly focus on detecting genetic risks from Tourette Disorder (TD) patients/families and conduct combined analysis across different disorders, including TD, ADHD, OCD, and ASD, to investigate the shared mechanisms to explain the high comorbidity in these patients.
FUN QUESTION: if you could be any animal, what would it be and why?
Cat, full of curiosity on everything. |